Transfected parvalbumin alters calcium homeostasis in teratocarcinoma PCC7 cells

Müller, B.K.; Kabos, P.; Belhage, B.; Neumann, T.; Kater, S.B.

European Journal of Cell Biology 69(4): 360-367

1996


ISSN/ISBN: 0171-9335
PMID: 8741218
Document Number: 455474
Indirect evidence supports a protective role of some EF-hand calcium-binding proteins against calcium-induced neurotoxicity. Little is known about how these proteins influence cytosolic calcium levels. After cloning the parvalbumin cDNA into an expression vector, teratocarcinoma cells (PCC7) were transfected. Parvalbumin-transfected and mock-transfected cells were loaded with the calcium indicator fura-2 and were exposed, in the same dish. to different concentrations of the calcium ionophore A23187 or to KCI. The results show that parvalbumin-transfected PCC7 cells had much better calcium buffering capacity than control cells.

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