Maternal anti-paternal reactivity--depends on etiology
Schütt, C.
Zentralblatt für Gynakologie 121(4): 202-205
1999
ISSN/ISBN: 0044-4197 PMID: 10355099 Document Number: 501822
Since TH1-cytokines compromise pregnancy and TH2-cytokines are produced at the maternal-fetal interface one can hypothesize that TH2-cytokines improve fetal survival. Cytotoxic T- or NK-cells are unable to recognize MHCI/II-negative trophoblast or become inactivated by HLA-G expression, respectively. Normal delivery at term might be assisted by a rapid reversal of the TH2 cytokine bias. Thus, the maternal immune state is most beneficial to reproductive fitness.
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